Preliminary scientific tests suggest conolidine could inhibit particular ion channels, reducing neuronal excitability and limiting discomfort alerts. This mechanism is especially relevant in neuropathic soreness, where by abnormal signaling triggers persistent distress. Additionally, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to discomfort percept... https://brucea703dzv3.wikimidpoint.com/user